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Review
. 2019 Jan;19(1):61-63.
doi: 10.7861/clinmedicine.19-1-61.

Cardiology: hypertrophic cardiomyopathy

Medical Masterclass contributors; John Firth
Review

Cardiology: hypertrophic cardiomyopathy

Medical Masterclass contributors et al. Clin Med (Lond). 2019 Jan.
No abstract available

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Figures

Fig 1.
Fig 1.
Effect of asymmetrical septal hypertrophy in HCM. In late systole the septum contracts down on the outflow tract, obstructing flow and generating a gradient. This generates a negative pressure (Venturi effect) just proximal to the obstruction, sucking the MV anteriorly (systolic anterior motion) and producing mitral regurgitation. Ao, aorta; LA, left atrium; LV, left ventricle; MV, mitral valve.
Fig 2.
Fig 2.
MRI of the heart in the short axis, showing asymmetrical hypertrophy of the interventricular septum in HCM (indicated by arrow). LV, left ventricular cavity; RV, right ventricular cavity.
Fig 3.
Fig 3.
Septal ablation in hypertrophic obstructive cardiomyopathy. (a) A wire is passed through a coronary guide catheter into the target septal artery, indicated by arrow. A balloon catheter is passed, the wire is removed and the balloon inflated to occlude the artery. (b) Dye is injected down the lumen of the balloon catheter into the distal septal artery to confirm correct positioning. (c) Absolute alcohol is then injected to destroy selectively the septal artery, leaving a stump. Simultaneous pressure recordings reveal a left ventricular outflow tract gradient (peak ventricular minus peak aortic pressure) of approximately 100 mmHg (d) before the procedure, falling to (e) 15 mmHg afterwards.
See this image and copyright information in PMC

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