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Q J Econ. Author manuscript; available in PMC 2009 Nov 16.
Published in final edited form as:
Q J Econ. 2009; 124(2): 597–636.
doi: 10.1162/qjec.2009.124.2.597
PMCID: PMC2777733
NIHMSID: NIHMS139186
PMID: 19920880

DOES MEDICARE SAVE LIVES?*

Associated Data

Supplementary Materials

Abstract

Health insurance characteristics shift at age 65 as most people become eligible for Medicare. We measure the impacts of these changes on patients who are admitted to hospitals through emergency departments for conditions with similar admission rates on weekdays and weekends. The age profiles of admissions and comorbidities for these patients are smooth at age 65, suggesting that the severity of illness is similar on either side of the Medicare threshold. In contrast, the number of procedures performed in hospitals and total list charges exhibit small but statistically significant discontinuities, implying that patients over 65 receive more services. We estimate a nearly 1-percentage-point drop in 7-day mortality for patients at age 65, equivalent to a 20% reduction in deaths for this severely ill patient group. The mortality gap persists for at least 9 months after admission.

I. Introduction

Medicare pays nearly one-fifth of total health care costs in the United States. Yet evidence on the health effects of the program is limited. Studies of aggregate death rates before and after the introduction of Medicare show little indication of a program impact (Finkelstein and McKnight 2005). The age profiles of mortality and self-reported health in the population as a whole are likewise remarkably smooth around the eligibility threshold at age 65 (Card, Dobkin, and Maestas 2004; Dow 2004). Although existing research has shown that the utilization of health care services increases once people become eligible for Medicare (e.g., Decker and Rapaport [2002], McWilliams et al. [2003, 2007], Card, Dobkin, and Maestas [2004]), the health impact of these additional services remains uncertain.

This paper presents new evidence on the health effects of Medicare, based on differences in mortality for severely ill people who are admitted to California hospitals just before and just after their 65th birthdays. Specifically, we focus on unplanned admissions through the emergency department (ED) for “non-deferrable” conditions—those with very similar weekend and weekday admission rates. These admissions include patients with diagnoses such as obstructive chronic bronchitis, acute myocardial infarction (AMI), and stroke and represent 12% of hospital admissions for people between the ages of 60 and 70, and 25% of all deaths between these ages. We argue that the decision to present at an ED for these conditions is unlikely to depend on insurance status. Consistent with this assertion, the arrival rate is essentially identical for patients just under and just over age 65. In contrast, admission rates for all causes jump 7% once people reach 65, and total ED admissions rise by 3%.

Focusing on nondeferrable admissions, we turn to an analysis of the age profiles of patient characteristics and outcomes, testing for discontinuities at age 65. The demographic composition and comorbidities of the sample trend smoothly through the age-65 barrier, as would be expected under the assumption of no differential sample selection before and after Medicare eligibility. On the other hand, the fraction of patients with Medicare as their primary insurer rises by about fifty percentage points, whereas the fraction with no insurance drops by eight percentage points.

Associated with these changes in insurance, we find a small but statistically significant increase in the number of procedures performed in the hospital and a 3% rise in total list charges. Using death records matched to our sample of hospital admissions, we find a clearly discernible drop in mortality once people become eligible for Medicare. Relative to people who are just under 65 when admitted, those who are just over 65 have about a 1% lower likelihood of death within a week of admission, or roughly a 20% reduction in 7-day mortality. A similar absolute reduction in mortality is registered at longer horizons and persists for at least 9 months, suggesting that the differential treatment afforded to those with Medicare coverage has an important impact on patient survival.

We conclude by discussing potential channels for the Medicare effect. One mechanism is an increase in services for the relatively small fraction (<10%) of patients who move from uninsured to insured status at age 65. Although previous studies have found evidence of this shift, our estimated mortality effects are too large to be driven entirely by gains for such a small group.1 The switch to Medicare coverage at 65 could also lead to increased treatments for patients who were previously covered by private insurance or Medicaid.2 Though we see modest rises in treatment intensity at age 65, it is unclear whether these changes account for the reduction in mortality, or whether other factors (such as changes in the timeliness of treatment) also play some role.

The next section presents a brief overview of the Medicare program and existing research on its impacts. Section III outlines our regression–discontinuity research design. Section IV describes our procedure for identifying nondeferrable ED admissions and summarizes our tests for differential selectivity between patients just under and just over 65. Section V presents our main analysis of the age profiles of treatment intensity and mortality for the subsample of nondeferrable admissions. Section VI discusses potential channels for the Medicare effect on treatment intensity and heath. Section VII concludes.

II. Medicare: Background and Previous Studies

II.A. Medicare Eligibility and Health Insurance

Medicare coverage is available to people who are 65 or older and have worked at least 10 years in covered employment.3 Medicare is also provided to people under 65 who are receiving Social Security Disability Insurance (DI): currently about 12% of the population are already on the program by the time they reach 65.4 Age-eligible individuals can enroll on the first day of the month that they turn 65 and obtain Medicare hospital insurance (Part A) for free. Medicare Part B, which covers doctor bills and some other charges, is available for a modest monthly premium.

The onset of Medicare eligibility leads to sharp changes in health insurance status at age 65. These changes are illustrated in Figure I, which presents data from the 1999–2003 National Health Interview Surveys (NHIS) on four different dimensions of insurance coverage: Medicare coverage; any insurance coverage; coverage by multiple policies; and having primary insurance coverage in a managed care policy. The figure shows the mean of each outcome by age (measured in quarters), as well as the fitted age profiles from a simple regression that includes a quadratic in age, a dummy for age over 65, and interactions of the dummy with age and age-squared.

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Changes in Health Insurance at Age 65 in National Health Interview Survey

Samples are based on data from the National Health Interview Survey, 1999–2003. The estimated discontinuities (and standard errors) at age 65 and the fitted lines are from a regression with a quadratic polynomial in age fully interacted with a dummy for age greater than or equal to 65. Models also include a dummy for people assigned to age 65.0 whose Medicare eligibility status is uncertain. Points represent means for people in each age cell (measured in quarters). Points for peope age 65.0 are not shown in the figure. Estimated fraction who have Medicare includes all forms of Medicare coverage—see text for discussion of underreporting of Medicare coverage in NHIS. Estimated fraction with multiple policies includes people with at least two forms of health insurance coverage (e.g., Medicare and private supplemental coverage, or Medicare and Medicaid).

The data show a 65-percentage-point rise in Medicare coverage at age 65, from 12% to just less than 80%. In fact, the jump may be even larger: a recent study by Cohen and Martinez (2007) analyzes the responses to a probe question added to the 2005 and later NHIS surveys and concludes that Medicare coverage for people over 65 is underreported by roughly eight percentage points in the basic NHIS.5 Associated with the rise in Medicare coverage is an increase of about nine percentage points in the fraction of people with any coverage, leaving only about three percent of the population over 65 uninsured, compared with about thirteen percent of those under 65.6

The other insurance characteristics shown in Figure I also change sharply at 65. The fraction of the population covered by multiple policies rises by about 45 percentage points, as many of those with private insurance before 65 obtain supplemental policies to “top up” their Medicare coverage.7 Conversely, the fraction of people covered by managed care in their primary policies falls by thirty percentage points. This drop reflects the relatively high rate of managed care coverage in the pre-65 insurance market, coupled with the relatively low fraction of Medicare recipients who chose managed care over traditional fee-for-service insurance in the period up to 2002.8

Overall, the data in Figure I show striking changes in the heath insurance coverage of the population at age 65. Within a few weeks of becoming eligible for Medicare, at least 80% of the population is enrolled in the program. In the process, about three-fourths of those who were previously uninsured obtain coverage. Many Medicare enrollees who were previously covered by private plans enroll in supplemental policies, creating a sharp rise in the incidence of multiple coverage. And, because most Medicare recipients choose traditional fee-for-service coverage, the fraction of the population with managed care is reduced by half.

II.B. Impacts of Medicare

Existing research has shown that the onset of Medicare age-eligibility leads to an increase in the use of health services. Two early studies focus on changes in the use of medical screening procedures by people who were less likely to have health insurance prior to 65. Decker and Rapaport (2002) find a relative increase in mammogram screenings for less educated and black women after 65. McWilliams et al. (2003) find that medical screenings increase more for people who lacked insurance coverage in the two years before reaching age 65. A study by Dow (2004) compares changes in hospitalization rates from 1963 (3 years before the introduction of Medicare) to 1970 (4 years after) for different age groups and finds a relative rise among those 65 and older. Card, Dobkin, and Maestas (2008) examine the age profiles of hospital admissions in California, Florida, and New York and find large increases in hospitalization rates at age 65, particularly for elective procedures such as coronary bypass surgery (16% increase in admission rates) and hip and knee replacement (23% increase). McWilliams et al. (2007) find that hospitalizations and doctor visits rise among previously uninsured individuals with hypertension, heart disease, diabetes, or stroke diagnosed before age 65.

As is true for health insurance more generally (see Levy and Meltzer [2004]), it has proven more difficult to identify the health impacts of Medicare.9 Most existing studies have focused on mortality as an indicator of health.10 An early study by Lichtenberg (2001) used Social Security Administration (SSA) life table data to test for a trend-break in the age profile of mortality at age 65. Although Lichtenberg identified a break, subsequent analysis by Dow (2004) showed that this is an artifact of the interval-smoothing procedure used to construct the SSA life tables. Comparisons based on unsmoothed data show no evidence of a shift at age 65 (Card, Dobkin, and Maestas 2004). Finkelstein and McKnight (2005) explore trends in state-specific mortality rates for people over 65 relative to those under 65, testing for a break around 1966—the year Medicare was introduced. They also examine the correlation between changes in relative mortality after 1966 and the fraction of elderly people in a region who were uninsured in 1963. Neither exercise suggests that the introduction of Medicare reduced the relative mortality of people over 65, though it should be noted that the power of these analyses is limited.

III. A Regression Discontinuity Analysis of Health Outcomes

As in earlier studies, we use comparisons across the age threshold for Medicare eligibility to measure the health impacts of the program. Unlike most existing studies, however, we attempt to isolate a subpopulation whose immediate mortality experience is more likely to be affected by differences in health care services provided to people once they are eligible for Medicare. Specifically, we focus on people who are admitted to the hospital through the ED for relatively severe illnesses. Any extra services (or improvement in the quality of services) offered to this subpopulation have at least a plausible chance of affecting short-run mortality. By comparison, Medicare-related changes in health care would have to have a very large impact on mortality to generate a detectable effect on the overall population.11

Our analysis is based on a reduced-form regression–discontinuity (RD) model of the form

yif(ai, α) + Post65i β + εi
(1)

where yi represents a health-related outcome for patient i, ai represents the patient’s age (measured in days from his or her 65th birthday), f (·) is a function that is continuous at age 65 with parameter vector α (e.g., a flexible polynomial), Post 65i is an indicator for whether the patient has passed his or her 65th birthday, and εi is an error term reflecting the influence of all other factors. In all specifications, we also interact the age polynomial with the Post 65i indicator to allow the slope of the age profile to vary on either side of the age 65 threshold. If yi is a measure of health care services provided to patient i, then we interpret β as a scaled estimate of the causal effect of Medicare coverage on the provision of services. As in other “fuzzy” RD designs (Hahn, Todd, and van de Klauuw 2001), the scale factor is just the difference in the probability of treatment on either side of the threshold, although in the case of Medicare, the treatment is potentially multidimensional (see Section VI, below).12 If yi is an indicator for mortality over some time horizon, then we interpret β as a scaled estimate of the causal effect of Medicare coverage on the likelihood of death in that time interval.

We defer a detailed discussion of the possible channels leading to the reduced form impact of Medicare coverage on health care services to Section VI. For now, we note that the data in Figure I suggest at least three alternatives: (1) an effect attributable to the increase in the overall fraction of the population with any health insurance; (2) an effect driven by people switching from an insurance carrier other than Medicare to a package that includes Medicare;13 and (3) an effect attributable to the change from managed care coverage to indemnity insurance. For example, hospitals may provide extra services if they know that a patient is covered by Medicare and supplemental insurance, rather than being uninsured, or covered by a typical pre-65 policy. Alternatively, there may be a reduction in the delay in verifying insurance status for Medicare patients or in receiving approval for certain procedures that are limited by managed care providers.

As emphasized by Lee (2008), the key assumption underlying an RD analysis is that assignment to either side of the discontinuity threshold (in our context, to being observed just a few weeks older or younger than 65) is as good as random. In the context of equation (1), this requires that

E[εi∣65 − δ < ai < 65] = E[εi∣65 ≤ ai < 65 + δ]
(2)

for δ sufficiently small, which ensures that a simple comparison of the mean of yi on either side of the age-65 threshold yields a consistent estimate of the parameter β.

In a sample of hospital admittees, the assumption that patients close to age 65 are “as good as randomly assigned” to either side of the age threshold may fail if insurance status affects the probability that a patient is admitted to the hospital. Because previous work has found that the onset of Medicare eligibility leads to an increase in hospitalization rates (Card, Dobkin, and Maestas 2008), this is a serious threat to an RD analysis of the health outcomes of patients. Figure II illustrates the difficulty in using counts of hospital admissions based on California discharge records from 1992 to 2002. (The sample is described more precisely below.) At age 65 the number of non-ED admissions jumps by approximately 12%, whereas the number of ED admissions rises by 3%. Assuming that the additional patients are not as sick as those who would enter the hospital regardless of Medicare eligibility, the average health of patients rises discretely at age 65.

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Number of Admissions by Route into Hospital, California, 1992–2002

The lines are fitted values from regressions that include a second-order polynomial in age fully interacted with a dummy for age ≧65 and a dummy variable for the month before people turn 65. The dependent variable is the log of the number of admissions by patient’s age (in days) at admission, for patients between 60 and 70 years of age. The count of admissions is based on hospital discharge records for California and includes admissions from January 1, 1992, to November 30, 2002. The points represent means of the dependent variable for 30-day cells. The age profile for unplanned ED admissions includes admissions that occurred through the emergency department and were unplanned. The category “Other Admissions” includes all other admissions.

In this paper we attempt to solve the sample selection problem by focusing on a subset of patients who are admitted through the ED for a relatively severe set of conditions that require immediate hospitalization. Specifically, we identify a set of admission diagnosis codes with similar ED admission rates on weekdays and weekends.14 We then test the assumption that there is no remaining selection bias associated with the age-65 boundary by looking for discontinuities in the number of admissions at 65 and the characteristics of patients on either side of the boundary. Importantly, our procedure for identifying an unselected sample is unrelated to the age of patients. Thus, our tests for selection bias are unaffected by “pretest bias” and provide a reasonable degree of confidence in the validity of our inferences.

As a check on inferences from this sample, we also use a simple bounding procedure (Horowitz and Manski 1995) to estimate a lower bound (in magnitude) for the impact of Medicare eligibility on other patient samples, including the overall population of hospital admissions. This bound is fairly tight because the relative size of the group of “extra” patients who only enter the hospital if they are over 65 is modest (at most 12%) and because the gap between actual mortality experience of all patients and the “worst case” bound for the extra patient group is small. For example, the average 28-day mortality rate of all people admitted to the hospital who are just over 65 is 4.6%, whereas the lower bound on the mortality of the extra patients is 0. As we discuss below, this means that the “worst case” bias created by the selective inflow of patients after 65 is −0.3%—a relatively small bias.

Even if there is no differential selection around the discontinuity threshold, inferences from an RD design can be compromised if there are other factors that change at the threshold. One concern is retirement: 65 is a traditional retirement age, and studies have shown that health is affected by employment status (Ruhm 2000). Nevertheless, we believe the confounding effect of retirement is relatively minor. First, as shown in Figure A in the Online Appendix, recent data show no discontinuity in the likelihood of working at exactly age 65.15 Second, the admission diagnoses included in our nondeferrable sample are relatively severe and would normally preclude an immediate return to work. But the mortality gap we observe in this sample at age 65 emerges within 7 days of initial admission to the hospital, and thus is unlikely to reflect differences in survival between people who return to work and those who do not.

Another concern with the age-65 threshold is that recommended medical practices may change at this age. Until recently, for example, U.S. government agencies recommended different influenza vaccination policies for people over and under 65 (Smith et al. 2006). Again, however, we think this is unlikely to affect the characteristics or treatment of patients admitted through the ED for nondeferrable conditions.

IV. Sample Construction and Validation

Our sample is drawn from the universe of records for patients discharged between January 1, 1992, and December 31, 2002 from hospitals regulated by the state of California. To be included in the sample, patients must have been admitted; thus, those who were sent home after treatment in the ED or who died either en route or in the ED do not appear in the sample.16 As explained in the Data Appendix (included in the Online Appendix to this paper), we drop discharge records for patients admitted before January 1, 1992, or on or after December 1, 2002, to avoid length-biased sampling problems.

The discharge data set includes basic patient information (month of discharge, age in days at the time of discharge, sex, race/ethnicity, and ZIP code of residence) as well as medical information, including the principal cause of admission,17 whether the admission was planned or unplanned, the route into the hospital (ED versus non-ED), the patient’s primary health insurance provider, the length of stay, and a list of all procedures performed in the hospital. It also includes a scrambled version of the patient’s Social Security Number, which can be used to track patients who are transferred or readmitted and to link mortality records. Appendix I describes our procedures for consolidating the records for patients who were transferred to new units in the same hospital, or to another hospital. It also describes the linked discharge-mortality file prepared for us by the California Department of Health Services, which we merge with the initial discharge file in order to determine the date of death for patients in the sample.

One limitation of the discharge data is that approximately 5% of 64-year-old patients in our sample have missing Social Security numbers (SSNs), compared with about 4% of those just over 65. Given that the in-hospital mortality rate of 60–64-year-old patients with missing SSNs is much higher than that of patients with valid SSNs (10.4% vs. 6.3%), we believe that the ability to match longer-term mortality outcomes for 1/5 of this group once they reach 65 will tend to bias down any observed mortality improvements at 65. In any case, in Section V.E., we present evidence showing that the mortality effects we estimate are unlikely to have arisen mechanically as a result of merging procedures or selectively missing data.

As discussed in the previous section, a critical step in our analysis is to select a subset of patients whose admission to the hospital is independent of insurance status. We do this by identifying a set of admission diagnosis codes (classified by 5-digit ICD-9) that have similar admission rates through the ED on weekdays and weekends. For example, if admission for a given diagnosis code were equally likely on a weekend and on a weekday, then weekend admissions should constitute 2/7 = 0.29 of total admissions for that diagnosis. We compute the fraction of weekend admissions for all ICD-9 codes and limit our analysis sample to those codes for which the t-statistic for a test that the fraction of weekend admissions = 2/7 is less than 0.965. Figure B in the Online Appendix shows how the distribution of the fraction of weekend admits over the diagnosis codes in our sample centers tightly around 2/7, in contrast to the distributions over all diagnosis codes and those associated with ED admissions (which center around a lower fraction of weekend admits). Arguably, these diagnoses are “nondeferrable,” and patients with these conditions will present at the ED at the same rate just before and just after their 65th birthdays, irrespective of Medicare coverage.

Table I summarizes the ten most common diagnosis codes in this subsample of 425,315 “nondeferrable” admissions. The largest diagnosis category is obstructive chronic bronchitis with acute exacerbation. (This includes patients with chronic obstructive pulmonary disease—a common diagnosis for smokers and ex-smokers.) Acute myocardial infarctions of all forms (i.e., including all 410.xx ICD-9 codes) represent the second most common group of admissions in our sample. The top ten diagnoses also include respiratory failure and two forms of stroke (intracerebral hemorrhage and cerebral artery occlusion).

TABLE I

Ten Most Common Diagnoses (ICD-9) in Nondeferrable Admissions Sample

Number of admissionsMean length of stayMean number of proceduresMean total list charges28-day death rate
Obstructive chronic bronchitis with acute exacerbation61,5586.21.226,0004.7
Respiratory failure24,32813.73.772,40022.5
AMI of other inferior wall (1st episode)21,1927.25.159,0006.9
AMI of other anterior wall (1st episode)15,7277.85.363,40010.6
Intracerebral hemorrhage10,75517.83.668,70029.6
Chronic airway obstruction, n.e.c.9,1026.51.522,1007.7
Fracture of neck of femur intertrochanteric section6,86814.22.744,1002.9
Cerebral artery occlusion, unspecified5,78215.23.733,9008.1
Convulsions unknown cause5,3385.11.222,8003.1
Asthma, unspecified with status asthmaticus5,0954.61.117,8000.9

Notes. Length of stay, procedure count, and hospital list charges are totals for all sequential hospital stays. Sample is drawn from California hospital discharge records for 1992–2002 for patients between the ages of 60 and 70 who were admitted to the hospital through the emergency department for an unplanned cause. Diagnoses are included only if a t-test for the equality of admission rates on weekends and weekdays has a t-statistic less than 0.965. List charges are in 2002 dollars.

To test that patients’ inclusion in the “nondeferrable” admissions subsample is independent of whether they are under or over 65, we conducted a regression discontinuity analysis of the count of admissions by age. This procedure is similar to the test of manipulation proposed by McCrary (2008), though we have a discrete running variable (age, measured in days) and we use a parametric rather than a nonparametric approach. Figure III shows the age profiles of the log of the daily admission count for four groups of ED admissions, based on the magnitude of the t-statistic for the test that the fraction of weekend admissions = 2/7. The groups of admission diagnoses with t-statistics in the top two quartiles (t > 6.62 and 2.54 < t < 6.62) show clear evidence of a jump at age 65, whereas the age profile for diagnoses in our preferred group, with |t| < 0.965, shows no visible evidence of an increase in admissions.

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Admissions through the ED by Quartile of t-test for Equality of Weekend and Weekday Admission Rates

See notes for Figure II. In this figure the population of patients with an unplanned admission through the ED is split into four groups based on the primary diagnosis ICD-9 code. Groups are defined by the range of the t-statistic for the test that 2/7 of admissions with the diagnosis occur on the weekend. The y-axis is the log of the number of admissions in the group by patient’s age (in days) at admission, for patients between 60 and 70 years of age. The count of admissions is based on hospital discharge records for California and includes admissions from January 1, 1992, to November 30, 2002.

Formal testing results are summarized in Table II. Each pair of columns in this table presents the estimated discontinuities at age 65 from two alternative RD models for the log of the number of admissions by age (in days) of the admitted patient. We limit the sample to people between the ages of 60 and 70, resulting in 3,652 observations—one for each potential value of age in days. Both specifications include a dummy for age over 65 and a quadratic polynomial fully interacted with the post-65 dummy. We have also fit the models with cubic polynomials and found no significant differences in the estimated values of the post-65 effects (see Table A in the Online Appendix).

TABLE II

Regression Discontinuity Models for Log (Number of Admissions) to California Hospitals by Age of Patient at Admission

Non-ED or planned
ED and unplanned
Weekend t-stat > 6.62
(1)(2)(3)(4)(5)(6)
Age over 65 (×100)11.9 (0.5)12.0 (0.5)2.4 (0.5)2.6 (0.5)3.2 (1.0)3.3 (1.1)
Dummy for just under 65NoYesNoYesNoYes
Weekend t-stat 2.54–6.62
Weekend t-stat 0.96–2.54
Weekend t-stat < 0.96
(7)(8)(9)(10)(11)(12)

Age over 65 (×100)3.6 (0.9)3.7 (1.0)2.7 (0.9)3.0 (1.0)0.6 (0.9)0.6 (0.9)
Dummy for just under 65NoYesNoYesNoYes

Notes. Standard errors in parentheses. Dependent variable in all models is the log of the number of admissions by patient’s age (in days) at admission, for patients between 60 and 70 years of age (3,652 observations). Count of admissions is based on hospital discharge records for California, and includes admissions from January 1, 1992, to November 30, 2002. All models include a second-order polynomial in age (in days) fully interacted with a dummy for age over 65. Models in even-numbered columns include a dummy for people who are within 1 month of their 65th birthdays, whose Medicare eligibility status at the time of admission is indeterminate. Sample in columns (1) and (2) includes admissions that either were planned or did not occur through the ED. Samples in all other columns include admissions that were unplanned and occurred through the ED. Samples in columns (5)–(12) are further restricted by including only admissions for diagnoses (ICD-9 classifications) for which the t-test for equality of weekend and weekday admission rates is in the range indicated. In columns (1)–(12) the coefficient on “age over 65” and its standard error have been multiplied by 100.

Although we know each patient’s exact age (measured in days) at the time of admission, we do not know patients’ birth-dates or the exact admission date.18 Because Medicare eligibility begins on the first day of the month that a person turns 65, people who are admitted in the period up to 31 days before reaching their 65th birthdays may or may not be eligible for Medicare. Figure C in the Online Appendix shows the fraction of admitted patients in our nondeferrable sample who are recorded as having Medicare as their primary insurance provider, by age in days for a narrow window around age 65. This fraction is relatively flat for people up to a month before their 65th birthdays, and then rises linearly in the 31 days before they reach age 65, as would be expected given Medicare eligibility rules and a uniform distribution of birthdates.

Because we do not know the Medicare eligibility status of patients who are admitted within 31 days of their 65th birthdays, the specifications reported in the even-numbered columns of Table II include a dummy for these observations. The addition of this dummy has relatively little impact on the estimated coefficients.19 Looking at the models in columns (1)–(4), we estimate that non-ED and planned ED admissions rise by about 12% at 65, whereas unplanned ED admissions rise by 2.6%. The remaining columns report the results for the four quartiles of unplanned ED admissions shown in Figure III. As suggested by the graph, the estimated models for our preferred subgroup of diagnoses (in columns (11) and (12)) show no evidence of a rise in admissions at age 65.

Although the number of admissions in our nondeferrable sample trends smoothly at age 65, we also tested for a change in the health characteristics of admissions at age 65. Specifically, we constructed a Charlson comorbidity score from the secondary diagnoses listed on each discharge record.20 Figure D in the Online Appendix shows the age profile in the Charlson comorbidity scores for the nondeferrable sample. There is no discernible evidence of a drop in the severity of comorbidities at age 65; in fact, formal RD analysis on the age profile indicates a small but statistically insignificant increase in severity (see Table B in the Online Appendix). If we interpret the rise as a true measure of the change in severity (and not a result of upcoding incentives in the Medicare payment system), it suggests that, if anything, our sample becomes slightly less healthy at age 65.

We have also checked for discontinuities in the case mix and demographic composition of the nondeferrable subsample at age 65. Counts of admissions for each primary diagnosis code included in our subsample trend smoothly with age, which suggests that the case mix is very stable through the age 65 boundary. Tests for jumps in the racial composition, sex, and fraction of Saturday or Sunday admissions (available on request) are all far below conventional critical values. To increase the power to detect differences in patient health, we used all the available covariates for an admission (including age, race/ethnicity, sex, year, month, and day of admission, and principal diagnosis fixed effects) to fit linear probability models for mortality over 7, 14, 28, 90, and 365 days. We then took the predicted mortality rates from these models and conducted an RD analysis, looking for any evidence that the predictors of mortality shift at age 65. The age profiles of 7-day and 28-day predicted mortality are shown in Figure E of the Online Appendix. (Results for other follow-up periods are very similar.) These age profiles are extremely smooth, and show no jump at age 65. In an RD specification with a quadratic in age and a dummy for over 65, interacted with the linear and quadratic terms, the t-statistics for the post-65 coefficient are 0.4 (7-day mortality) and 0.25 (28-day mortality), providing no evidence that the observable health of the sample changes at age 65.

A final piece of indirect evidence that patients with severe conditions are not more (or less) likely to present at the ED once they become Medicare-eligible comes from studies of the effect of cost-sharing (i.e., copayments and deductibles) on the use of the ED.21 In the RAND health insurance experiment, patients with cost-sharing insurance plans were no less likely to present at the ED for the most serious conditions (chest pain/acute heart disease, surgical abdominal disease, acute eye injuries, second-degree burns) than those with the “free” plan (O’Grady et al. 1985). Similarly, two recent studies of the introduction of cost-sharing for ED visits by patients in a health maintenance organization (HMO) (Selby, Fireman, and Swain 1996; Wharam et al. 2007) conclude that copayments have no large or significant effect on ED visits for patients with severe conditions. The sample sizes in these studies are modest, however, and one certainly could not detect changes on the order of 3% in the use of the ED, which is the magnitude of the jump at age 65 for all unplanned ED admissions in the California discharge data. A fourth HMO-based study by Hsu et al. (2006) has much larger sample sizes and concludes that the introduction of modest copayment requirements reduces ED visits. This study does not break down ED visits by the severity of patients’ conditions, though the authors find that patients with copayment requirements have somewhat better clinical outcomes, which suggests that patients with life-threatening conditions such as AMI, stroke, and chronic obstructive pulmonary disease are obtaining appropriate medical services. Overall we interpret these studies as supporting the hypothesis that people with severe conditions present at the ED independent of their insurance status.

V. Shifts in Insurance, Health Services, and Mortality at 65

V.A. Insurance

We now turn to the impact of the Medicare eligibility age on health-related outcomes. We begin by looking at health insurance coverage. Figure IV shows the age profiles of the fractions of people with various primary insurers (private, Medicaid, Medicare, other, and none) in the nondeferrable admissions subsample. Consistent with the patterns in Figure I for the overall population, we see a big increase in the fraction of patients with Medicare as their primary insurer at 65, coupled with a decline in the fraction with no insurance. RD models for health insurance outcomes are presented in Table III. The models follow the same specification as in Table II, although we now include a set of covariates (year, month and day of admission, race/ethnicity, sex, and admission diagnosis fixed effects) in the specifications shown in even-numbered columns. For reference, the specifications in the odd-numbered columns exclude these controls and also exclude the dummy for admissions in the 31 days before a patient’s 65th birthday.

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Primary Insurance Coverage of Admitted Patients

See notes for Figure II. In this figure the y-axis represents the fraction of patients with different classes of primary insurance coverage. Sample includes 425,315 patients with nondeferrable primary diagnoses, defined as unplanned admissions through the emergency department for diagnoses with a t-statistic for the test of equal weekday and weekend admission rates of 0.965 or less. Medicare eligibility status of patients within one month of their 65th birthdays is uncertain and we have excluded these observations.

TABLE III

Regression Discontinuity Models for Probability of Different Forms of Primary Insurance Coverage

Medicare
Private
Medicaid
Uninsured
(1)(2)(3)(4)(5)(6)(7)(8)
Age over 65 (×100)43.9 (0.4)47.5 (0.4)−24.8 (0.4)−26.8 (0.4)−10.1 (0.3)−10.8 (0.3)−7.4 (0.2)−8.0 (0.2)
Additional controlsNoYesNoYesNoYesNoYes
Mean of dependent variable for patients aged 64–65 (×100)24.043.343.39.7

Notes. Standard errors in parentheses. Dependent variable is indicator for type of insurance listed as “primary insurer” on discharge record. Sample includes 425,315 observations on patients between the ages of 60 and 70 admitted to California hospitals between January 1, 1992, and November 30, 2002 for an unplanned admission through the emergency department, with a diagnosis (ICD-9) for which the t-test for equality of weekend and weekday admission rates is less than 0.96 in absolute value. All models include second-order polynomial in age (in days) fully interacted with dummy for age over 65 and are fit by OLS. Models in even-numbered columns include the following additional controls: a dummy for people who are within one month of their 65th birthday; dummies for month, year, sex, race/ethnicity, and admission on Saturday or Sunday; and a complete set of unrestricted fixed effects for each ICD-9 admission diagnosis. In columns (1)–(8) the coefficient on “age over 65” and its standard error have been multiplied by 100.

The regression results confirm the visual impressions conveyed in Figure IV. At age 65, the fraction of patients with Medicare as their primary insurer rises by about 47 percentage points, whereas the fractions with private insurance and Medicaid both fall.22 Note that in the sample of nondeferrable admissions the Medicare coverage rate at age 64 is 24%, substantially higher than in the overall population (shown in Figure I). Presumably this reflects the fact that many of these patients are chronically ill and on DI prior to 65. The percentage with no insurance at 64 is correspondingly a little lower than in the overall population (10% versus about 13%), and the reduction in the rate of noninsurance at 65 is a little smaller (−8 percentage points in the nondeferrable subsample, versus −9.5 percentage points for the population as a whole). Nevertheless, as in the population as a whole, patients with nondeferrable conditions have much different insurance coverage just after age 65 than just before.

V.B. Intensity of Treatment

We have three basic measures of the intensity of treatment offered to patients: the length of their stays in hospital, the number of procedures performed, and total hospital list charges.23 Figure V shows the age profiles for these measures, whereas Table IV presents RD models similar to the specifications in Table III. The age profile for mean length of stay is somewhat noisier than the other two profiles, but all three profiles suggest an upward jump at 65. The estimation results in Table IV show that mean length of stay increases by 0.4 days (or about 4.5%) at 65, though the estimated gain is not statistically significant. Similarly, the number of procedures jumps by 0.1, or approximately 4% (with a t-ratio around 4), whereas log list charges jump by 2.5% (with a t-ratio of around 2.6).

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Three Measures of Inpatient Treatment Intensity

See notes to Figure IV. Sample includes unplanned admissions through the emergency department for diagnoses with a t-statistic for the test of equal weekday and weekend admission rates of 0.965 or less. In this figure the sample is further restricted to patients with valid SSNs (407,386 observations). Sample for log list charges excludes patients admitted to Kaiser hospitals. Length of stay, number of procedures, and list charges are cumulated over all consecutive hospitalizations. List charges are measured in 2002 dollars.

TABLE IV

Regression Discontinuity Models for Changes in Treatment Intensity

Length of stay (days)
Number of procedures
Log list charges (×100)
(1)(2)(3)(4)(5)(6)
Age over 650.37 (0.24)0.35 (0.26)0.09 (0.03)0.11 (0.03)2.5 (1.1)2.6 (1.0)
Additional controlsNoYesNoYesNoYes
Mean of dependent variable for patients aged 63 or 648.122.509.87

Notes. Standard errors in parentheses. Dependent variable is length of stay in days (columns (1) and (2)), number of procedures performed (columns (3) and (4)), and log of total list charges (columns (5) and (6)). Sample includes 407,386 (352,652 in columns (5) and (6)) observations on patients with valid SSNs between the ages of 60 and 70 admitted to California hospitals between January 1, 1992, and November 30, 2002 for an unplanned admission through the ED. Data on list charges are missing for Kaiser hospitals. See note to Table III for additional details on sample, and list of additional covariates included in even-numbered columns. In columns (5) and (6) the coefficient on “age over 65” and its standard error have been multiplied by 100.

One concern with an RD model for the logarithm of list charges is that the dispersion in charges may increase (or decrease) once patients become Medicare-eligible. If this is true, then the expected level of charges may rise by more (or less) than 3% at age 65 (see Manning [1998] for a general discussion of modeling health spending). To evaluate this concern we fit RD models to the standard deviation of log list charges for patients in each age-at-admission cell (with age measured in days). These models showed no large or statistically significant effect on the dispersion in list charges at age 65. We also fit RD models to the 75th and 90th percentiles of list charges for each age-at-admission cell. These models showed that both the 75th and 90th percentiles of list charges increase by about 2%–3% at age 65. (The regression models, and associated graphs, are presented in the Online Appendix, Table C and Figures F and G.)

Overall, we conclude that there are modest but statistically significant increases in the intensity of treatment at age 65 for patients in our nondeferrable admissions sample, on the order of 3%. These increases are much smaller than the 10%–25% increases in rates of elective procedures such as hip and knee replacements observed in the overall population (Card, Dobkin, and Maestas 2008) but suggest that the availability of Medicare affects the utilization of health care services even for severely ill patients.

We also performed a more detailed analysis of the changes in the use of specific procedures at age 65 for two major sets of diagnoses: obstructive chronic bronchitis with acute exacerbation (the largest ICD-9 in our nondeferrable sample, shown in row (1) of Table I) and acute myocardial infarction (AMI), which combines the various detailed AMI diagnoses in our nondeferrable sample. The results are summarized in Table D of the Online Appendix. For AMI admissions we see a relatively large and precisely estimated increase in the overall number of procedures at age 65 (a rise of 0.44 on a base rate of 5.0 among 64-year-olds, or approximately 9%) and significant increases in the use of several important diagnostic procedures, including coronary arteriography, cardiac catheterization, and angiocardiography.24 In contrast, for obstructive chronic bronchitis patients, we see no change in the overall number of procedures and small increases or decreases in the incidence of specific procedures. This analysis suggests that the relatively small increase in the overall number of procedures for all admission diagnoses in Table IV is masking larger increases for certain “procedure-intensive” diagnoses, such as AMI, and near-constancy for other diagnoses. Unfortunately, the sample sizes for other diagnoses are too small to permit a more extensive investigation. We conclude, however, that the onset of Medicare eligibility is associated with an increase in the use of specific potentially life-saving procedures.

V.C. Transfers and Readmissions

Patients who are initially admitted for acute care may be transferred (i.e., discharged and immediately readmitted) to another care/treatment unit in the same hospital, to another hospital, or to nonhospital care (e.g., a nursing home).25 Because our data are derived from hospital discharge records, we cannot measure transfers to standalone skilled nursing facilities or to other care options that may be substitutable for postacute care in a hospital setting. Nevertheless, we find that within- and between-hospital transfer rates rise at age 65, with a particularly large rise in within-hospital transfers (25%) that appears to be driven by a jump in the rate of transfer to skilled nursing facilities within the same hospital. We also find a marginally significant reduction in the probability that patients are readmitted to a California hospital within 28 days after their initial admission (point estimate = −0.6-percentage-point reduction on a base readmission rate of 17.0% for 64-year-olds, t = 1.70). Graphs and estimates for transfers and readmissions are presented in the Online Appendix (Figures H and I and Table E).

V.D. Mortality

Figure VI plots the age profiles for the probability of death within 7, 14, 28, 90, 180, and 365 days of admission to the hospital, whereas the first two rows of Table V present estimates from RD regression models corresponding to each of these outcomes. Inspection of Figure VI shows that each of the mortality measures has a drop on the order of 1.0 percentage point at age 65. The regression estimates in Table V confirm this: we observe a reduction in 7-day mortality of about 0.7 to 1.0 percentage points that persists over the longer follow-up periods. The effect is relatively precisely measured in the shortest time intervals but has an increasing sampling error as the follow-up window is extended, yielding t-ratios of about 5 at 7 days, about 3 at 28 days, and around 1.8 at 365 days.

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Patient Mortality Rates over Different Follow-Up Intervals

See notes to Figure IV. Sample includes unplanned admissions through the emergency department for diagnoses with a t-statistic for the test of equal weekday and weekend admission rates of 0.965 or less. In this figure the sample is further restricted to patients with valid SSNs (407,386 observations). Deaths include include in-hospital and out-of-hospital deaths.

TABLE V

Regression Discontinuity Estimates of Changes in Mortality Rates

Death rate in
7 days14 days28 days90 days180 days365 days
Estimated discontinuity at age 65 (×100)
Fully interacted quadratic with no additional controls−1.1 (0.2)−1.0 (0.2)−1.1 (0.3)−1.1 (0.3)−1.2 (0.4)−1.0 (0.4)
Fully interacted quadratic plus additional controls−1.0 (0.2)−0.8 (0.2)−0.9 (0.3)−0.9 (0.3)−0.8 (0.3)−0.7 (0.4)
Fully interacted cubic plus additional controls−0.7 (0.3)−0.7 (0.2)−0.6 (0.4)−0.9 (0.4)−0.9 (0.5)−0.4 (0.5)
Local linear regression procedure fit separately to left and right with rule-of-thumb bandwidths−0.8 (0.2)−0.8 (0.2)−0.8 (0.2)−0.9 (0.2)−1.1 (0.3)−0.8 (0.3)
Mean of dependent variable (%)5.17.19.814.718.423.0

Notes. Standard errors in parentheses. Dependent variable is indicator for death within interval indicated by column heading. Entries in rows (1)–(3) are estimated coefficients of dummy for age over 65 from models that include a quadratic polynomial in age (rows (1) and (2)) or a cubic polynomial in age (row (3)) fully interacted with a dummy for age over 65. Models in rows (2) and (3) include the following additional controls: a dummy for people who are within 1 month of their 65 birthdays, dummies for year, month, sex, race/ethnicity, and Saturday or Sunday admissions, and unrestricted fixed effects for each ICD-9 admission diagnosis. Entries in row (4) are estimated discontinuities from a local linear regression procedure, fit separately to the left and right, with independently selected bandwidths from a rule-of-thumb procedure suggested by Fan and Gijbels (1996). Sample includes 407,386 observations on patients between the ages of 60 and 70 admitted to California hospitals between January 1, 1992, and November 30, 2002, for unplanned admission through the ED who have nonmissing Social Security numbers. All coefficients and their SEs have been multiplied by 100.

We have performed extensive robustness checks to ensure that the mortality results are not an artifact of a particular specification of the RD model. As shown in row (3) of Table V, specifications with a cubic age polynomial yield estimates that are similar to the simpler quadratic models, though typically a little smaller, particularly for the 28-day follow-up window. (Figure J in the Online Appendix compares the fits of linear, quadratic, and cubic RD models for 28-day mortality.) We also refit the models using logits (rather than linear probability specifications) and obtained essentially identical estimates of the change in the probability of death at age 65 (see Table F of the Online Appendix). Finally, we used local linear regression (LLR) models to obtain “nonparametric” estimates of the mortality rates for patients just under and just over 65 (Hahn, Todd, and van der Klaauw 2001; Imbens and Lemieux 2008). Row (4) of Table V presents LLR-based estimates using a triangular kernel and the rule-of-thumb bandwidth selection procedure suggested by Fan and Gijbels (1996). These are very similar to the parametric estimates using a quadratic polynomial but a little more precise. Figure K of the Online Appendix presents LLR-based estimates of the 28-day mortality rate for patients on each side of the age 65 boundary, using all possible bandwidths between 1 month and 5 years.26 The estimated mortality rates for patients just under 65 and patients just over 65 stabilize once the bandwidth reaches about 1 year, centering on values close to the predicted values from our basic quadratic specification.

We used the specification from row (2) of Table V to fit parametric RD models for the probability of death in all possible follow-up windows between 1 day and 2 years. The resulting estimates of the jump in mortality at age 65 are plotted in Figure VII, along with the associated 95% confidence intervals. The data show a robust pattern of reductions in mortality for windows of up to 9 months on the order of 0.8–1.0 percentage points, with somewhat smaller point estimates for windows of 1–2 years.

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Estimates of the Discontinuity in Mortality Rates at Age 65 over Various Follow-Up Periods

See notes to Figure VI. The estimates in this figure are from a regression with a quadratic polynomial in age fully interacted with a dummy for age over 65. The regressions also include a dummy for patients within one month of their 65th birthdays, month and year dummies, fixed effects for the primary diagnosis, and dummies for race, sex, and admissions on Saturday or Sunday. Regression discontinuities are estimated for probability of death within 7, 14, and 28 days and then at 30-day intervals. The estimates and associated 95% confidence intervals from each regression are then linearly interpolated in the figure.

Our estimates of the mortality effect of Medicare eligibility are relatively large: they represent a 14%–20% reduction in 7-day mortality, a 7%–9% reduction in 28-day mortality, and a 2%–4% reduction in 1-year mortality relative to death rates among 64-year-olds with similar conditions at admission. The emergence of the effect within 7 days of admission suggests that the extra services or changes in the quality of services provided to Medicare-eligible patients have an immediate life-saving impact.

It is also worth noting that the mortality reductions estimated in Table V appear to reflect changes in the treatment of patients with Medicare within the same hospital, rather than patient sorting to higher-quality hospitals at 65. The fractions of patients with nondeferrable conditions entering different kinds of hospitals show only small changes at age 65. The largest change is a reduction of about three percentage points in the fraction entering county hospitals. Interestingly, the 28-day mortality rate for 63–64-year-olds is actually lower at county hospitals (6.8%) than at nonprofit (9.2%), for-profit (9.0%), or district (9.7%) hospitals in our data, so it is implausible that such a small shift in patients out of county hospitals could have much affect on average mortality.27 Thus, it does not appear that Medicare reduces mortality by shifting patients to better hospitals.

V.E. Robustness of Mortality Estimates—A Bounding Approach

To further probe our estimated mortality effects, we used a simple bounding procedure to obtain lower-bound estimates of the (absolute) mortality effect of Medicare eligibility on broader samples of hospital admissions, including the entire patient population. The basis of this procedure is the observation that in any sample of sick people close to age 65 there are two subgroups: one group (which we index with subscript 1) who enter the hospital regardless of whether they are Medicare-eligible or not; and a second group (indexed by subscript 2) who will only enter the hospital if they are over 65. Let α ≥ 0 represent the sample fraction of the second group. We have argued that among people with non-deferrable conditions, α = 0. In more general patient populations, however, α > 0, and a comparison of mortality between patients just over and just under 65 contains a selectivity bias.

Let m1 denote the mortality rate of the first group if they enter the hospital just before their 65th birthdays and let m1 denote the mortality rate if they enter after 65. The causal effect of Medicare eligibility for group 1 is Δ=m1m1. The observed mortality rate of the patient population just over 65 is an average for groups 1 and 2,

m¯=(1α)m1+αm2=(1α)(m1+Δ)+αm2,

where m2 is the post-65 mortality rate of group 2. Using this expression it is easy to show that

m¯m1=Δα/(1α)×(m¯m2).
(3)

Thus, the mortality differential between the post-65 patient population and the pre-65 patient population is equal to Δ, the causal effect of Medicare eligibility on group 1, plus a bias term,

Bias=α/(1α)×(m¯m2),

which depends on the fraction of group 2 and the deviation of their post-65 mortality rate from the average of groups 1 and 2. Because m2 > 0, a lower bound on the absolute value of the bias caused by the presence of group 2 in the post-65 patient population is

WorstcaseBias=α/(1α)×m¯.
(4)

This bias tends to 0 as α →0, and is proportional to m̄.

Table VI presents estimates of the various terms in equation (3) for the 28-day mortality rates of various patient populations, including all patients (column (1)); those who enter the hospital via a route other than the ED, or for a planned hospitalization (which we call “elective” admissions, in column (2)); those who enter via the ED for an unplanned hospitalization (column (3)); and the four subgroups of the unplanned-ED group, based on admission diagnoses with different ranges of weekend versus weekday admissions (i.e., the four subgroups graphed in Figure III) in columns (4)–(7). The first row of Table VI presents the estimated RD in the log of the number of hospital admissions at age 65, which is an estimate of α/(1 − α).28 Row (2) shows the estimated change in the mortality rate of patients at age 65 (i.e., the estimate of m̄m1), obtained from an RD model with an interacted quadratic function of age fit to aggregated mortality rates by age in days.29 Row (3) shows an estimate of the constant in the mortality regression, which is our estimate of the mortality rate for people just under 65. (The implied estimate of m̄ is therefore the sum of the entries in rows (2) and (3).) Row (4) shows our estimate of the worst-case selectivity bias, based on equation (4), whereas row (5) shows our lower bound estimate of the effect of Medicare eligibility on the patient population, and row (6) shows an estimated sampling error for this bound. Finally, for reference, row (7) shows the fraction of patients in each subgroup.

TABLE VI

Estimated Lower Bounds for Change in 28-Day Mortality at Age 65

Unplanned ED admissions (by range of t-test for weekend admission rate)
All admissions (1)Planned or non-ED admissions (2)Unplanned ED admissions (3)t > 6.62 (4)2.54 < t < 6.51 (5)0.96 < t < 2.54 (6)t < 0.96 (7)
Estimated discontinuity in log of number of admissions at age 65 (×100)7.1211.852.433.233.572.700.56
Estimated discontinuity in probability of death at age 65 (×100)−0.46−0.29−0.490.27−0.63−0.45−1.09
Mean probability of death for people aged 64 (×100)5.103.296.862.687.416.8710.25
Estimated “worst case” bound on selection bias component of change in death rate at age 65−0.33−0.36−0.15−0.10−0.24−0.17−0.05
Estimated lower bound for causal effect of reaching age 65 on death rate (standard error)−0.13 (0.07)0.06 (0.09)−0.33 (0.12)0.37 (0.16)−0.39 (0.25)−0.27 (0.24)−1.04 (0.29)
Share of admissions in column sample1.000.500.500.110.130.120.12

Notes. Row (1) presents estimated regression discontinuities in the log of the number of hospital admissions at age 65, from a specification fit to admission counts by age for patients age 60 to 70 (3,652 observations) that includes a quadratic polynomial in age, fully interacted with a dummy for age over 65. Row (2) presents estimated regression discontinuities in the probability of death at age 65 from a specification fit to mean death rates by age at admission data for patients age 60 to 70 (3,652 observations) that includes a quadratic polynomial in age, fully interacted with a dummy for age over 65. Row (3) presents estimated mean probabilities of death within 28 days of admission for patients who are 64 years old. Row (4) presents the estimated worst case bound on the selectivity component of the change in death rates at age 65, computed as − row (1) × (row (2) + row (3))/100. Row (5) is the estimated lower bound on the causal effect of reaching age 65 on the 28-day death rate, computed as row (8) − row (4). The standard error is computed by the delta method, using the sampling errors of the estimates of the entries in rows (1), (2), and (3), and their sampling covariances. In rows (1)–(3) the coefficients have been multiplied by 100.

Three key conclusions emerge from the table. First, the lower-bound estimate of the overall effect of Medicare on the 28-day death rate of the entire patient population is −0.13% (and only marginally significant). This is about 1/10 as large as our estimate of the effect on the nondeferrable admissions group, who represent 12% of the overall patient population. Second, for “elective” admissions (column (2)), our point estimate of the lower bound mortality effect is actually positive (as it is for the top quartile of diagnoses with lowest weekend admission rates in column (5)). For these admissions we cannot rule out the possibility that selection bias explains the entire (relatively small) drop in mortality we see after age 65. Even for the two middle quartiles of weekend/weekday admission codes, the estimated lower bounds on the Medicare effect are small. Thus, virtually all of the (lower bound) mortality effect we observe for the overall patient sample is attributable to the reduction in mortality for the nondeferrable subgroup.

A third observation is that the unadjusted change in mortality at age 65 for the top quartile diagnosis group (column (4)) is actually positive (+0.27%). This is reassuring in two ways. First, it proves that there is no mechanical data problem that is causing us to measure lower death rates for all patients over 65.30 Second, the diagnoses in this quartile are relatively non-life-threatening. In particular, the 28-day mortality rate for 64-year-old patients in this group is only 2.7%, somewhat below the death rate for patients admitted on an elective basis. It would be surprising if Medicare eligibility had much effect on mortality for such a relatively healthy group, and the estimates imply that it does not.

VI. Discussion

Our empirical results point to a significant positive effect of Medicare eligibility on the intensity of treatment for acutely ill patients with nondeferrable conditions and a negative effect on patient mortality. In this section we discuss the possible channels for this effect. To aid in this discussion, it is helpful to consider a simplified causal model in which Medicare eligibility affects insurance characteristics, insurance affects health care services, and health services affect mortality. Building on the analysis in Section II, suppose that patient i has a health insurance package with a vector of characteristics zi, including whether i has any coverage, whether he or she has Medicare or some other form of primary coverage, and (possibly) other characteristics. Assume that the age profile for zi is generated by a model of the form

zig(ai, γz) + Post65iπ + υzi
(5)

where g is a smooth function of age (ai) with parameters γz, υzi is an error term that is mean-independent of the dummy Post65i, and π represents the vector of discontinuities in insurance characteristics at age 65. Suppose that the quality-adjusted health care services delivered to patient i (Si) depend on age, an error term υsi, and the characteristics of the insurance package:31

Sih(ai, γs) + θzi + υsi.
(6)

Finally, assume that the likelihood of death of patient i (yi = 1) depends on age and on quality-adjusted health services:

yik(ai, γs) + λSi + υyi.
(7)

Equations (5), (6), and (7) yield reduced-form models such as equation (1), with a discontinuity in health care services at age 65 equal to

βs = θπ
(8a)

and a discontinuity in mortality equal to

βy = λθπ.
(8b)

In this simplified setup, each element of the insurance package represents a separate “channel” that contributes additively to the reduced form effects on services and mortality. For example, the kth element of zi contributes θkπk to the RD in services and λθkπk to the RD in mortality. Unfortunately, we have no information on the individual components of θ, and only limited information on the vector π of insurance changes at age 65. For example, we do not observe secondary coverage, or whether the primary insurance is managed care. Nevertheless, it is possible to shed some light on the mortality effect associated with one key insurance characteristic: whether the patient has any insurance coverage or none.

In particular, note that the maximum contribution of the “any coverage” channel cannot exceed πc (the jump in coverage at 65) times the average mortality rate of uninsured 64-year-olds, because the extension of coverage to the previously uninsured group can only reduce their mortality rate to 0. The average 7-day mortality rate of uninsured patients who are just under 65 years of age in our nondeferrable admission subsample is 0.05, whereas πc = 0.08 (Table III, column (8)). Thus the maximum reduction in mortality attributable to the reduction in the number of people with no health insurance is 0.004—about 40% of the 7-day mortality effect we estimate. This is an extreme bound because it is based on the assumption that none of the previously uninsured would die if they were covered. A more plausible bound is that insurance coverage reduces the death rate by no more than one-half: in this case the “any coverage” channel can explain at most 20% of the total mortality effect.

In principle we can gain some additional insight by comparing changes in health insurance, the intensity of treatment, and mortality for different subgroups of patients.32 Unfortunately, the limited demographic variables in our discharge data make this a challenging exercise. Comparisons across race/ethnicity groups are uninformative, because the sample sizes for blacks (n = 41,000) and Hispanics (n = 66,280) are too small to obtain useful estimates. We also tried dividing patients into two groups based on the average fraction of 55–64-year-old patients from the same ZIP code who had no insurance coverage. Even here, we were unable to estimate systematic differences in the changes in treatment intensity or mortality outcomes at age 65 between residents from “low-insurance” and “high-insurance” ZIP codes. We do find significant increases in the numbers of procedures and significant reductions in mortality even for patients from the high-insurance ZIP codes, suggesting that an increase in insurance coverage per se is not the explanation for the impacts of Medicare.

An alternative explanation for the measured mortality effects is that for most people Medicare imposes fewer restrictions than private insurance or Medicaid, leading to more (and possibly higher-quality) services to patients over 65 than to those under 65.33 Card, Dobkin, and Maestas (2008) find clear evidence of this mechanism for a wide range of nonurgent medical procedures, such as surgery to insert a stent in a blocked coronary artery (which rises by 11% at age 65 in California, Florida, and New York), hip and knee replacement surgery (which rises by 23%), and gall bladder removal surgery (which rises by 18%). In fact, Table IV presents evidence of small increases (3%–4%) in the number of procedures and in total list charges at age 65 for patients with nondeferrable conditions. Arguably, however, such small increases in the intensity of treatment are unlikely to generate a one-percentage-point reduction in mortality, though as we noted above, the increase in procedures for AMI cases—which may be more sensitive to medical intervention—is closer to 10%.34 Thus, the precise mechanisms for the mortality effect remain unclear, though we believe the evidence points to a combination of channels.

VII. Summary and Conclusions

A longstanding question in health economics is whether health insurance affects health. This question is particularly relevant for Medicare, the largest medical insurance program in the country, which provides nearly universal coverage to people once they turn 65. We focus on measuring the health effects of Medicare eligibility for a relatively sick population—specifically, people who are admitted to hospitals through the ED with diagnoses that have similar admission rates on weekdays and weekends. In contrast to elective hospitalizations, there is no jump in these “nondeferrable” hospital admissions at age 65. Moreover, the predicted mortality rate of admitted patients (based on demographics and admission diagnoses) trends smoothly. These findings suggest that the underlying health of patients admitted with nondeferrable conditions is very similar whether the patients are just under or just over 65.

In light of this conclusion, we use a regression discontinuity approach to measure the impacts of reaching age 65 on the intensity of treatment in the hospital, and on mortality for up to 2 years after the hospital admission. We find modest but statistically significant increases in measures of treatment intensity at age 65, including the number of procedures performed in the hospital and total list charges. Associated with these changes, we find an important and large reduction in patient mortality at age 65. Medicare eligibility reduces 7-day mortality by about 0.8 to 1.0 percentage points, with similar-sized and statistically significant reductions at windows of up to 9 months. We probe the robustness of these findings by using a bounding procedure to evaluate the lower-bound effect of Medicare eligibility on the entire hospital patient population. The bounds for the overall population are consistent with the magnitude of the effect we estimate for patients with nondeferrable conditions, providing further credence to our basic results.

The magnitude of the estimated mortality effect of Medicare eligibility is too large to be driven solely by changes among the 8% of the patient population who move from no health insurance coverage to Medicare when they reach 65. This is an important distinction between our analysis and existing studies that have attributed much larger mortality gains to insurance status in specialized populations such as auto accident victims (Doyle 2005) and former Medi-Cal recipients (Lurie et al. 1984, 1986). Instead, our findings point to a more widespread effect of Medicare, including an impact on patients who were insured prior to 65. Given the relatively modest increases in the intensity of treatment we measure at age 65, however, we conclude that the actual mechanism for this effect is unclear.

An important limitation of our analysis is that it focuses on just one health outcome, albeit an important one. Certainly, Medicare might affect other dimensions of health and other patient populations, with effects that arise through channels other than hospitalization (e.g., outpatient care and prescription drug use) and that persist over a longer time horizon than that supported by our research design. Nonetheless, our analysis illustrates an important lesson for future research. Any plausible effect of insurance on health status in the general population will likely be small and easily confounded by selection effects in observational settings. Indeed, the only randomized health insurance experiment ever mounted found insignificant impacts of insurance on the health status of the overall population (Newhouse 1993). Further progress on this question will require research designs based on samples larger than those typically available for health services research, along with particular attentiveness to the selection problem.

Supplementary Material

Appendix

Appendix I

We use annual hospital discharge files from the state of California for the period 1992–2002. These files represent a census of discharges from state-regulated acute care hospitals, collected by OSHPD, the Office of Statewide Health Planning and Development, under the California Code of Regulations, Title 22. The data set excludes people admitted to federally regulated hospitals such as VA hospitals. Our working sample includes people admitted to the hospital between January 1, 1992, and November 30, 2002, whose age at admission was between 60 and 70. The discharge files include the patient’s sex, race/ethnicity, ZIP code of residence, date of admission, source of admission, route of admission (ED or not), type of admission (scheduled or unscheduled), principal diagnosis, other diagnoses present at admission, a list of procedures performed, total list charges, expected payer code, disposition of patient (routine discharge, transfer to another hospital, etc.), and date of discharge. We obtained a restricted use version of the file that also includes the patient’s age in days at admission (calculated from the exact date of admission and exact date of birth). The file also includes a unique patient identifier and an indicator for whether the patient provided a valid SSN.

We also obtained from OSHPD a separate file constructed by OSHPD by matching individuals in the discharge database to state records of death, by name/SSN/date of birth. We merged this file with the discharge record file by patient identifier.

In our analysis we combine all consecutive hospital admissions into a single record defined by the first date of admission (and by the descriptors associated with this first admission). We cumulate the length of stay, the number of procedures, and list charges across all stays.

Footnotes

1Canto et al. (2000) and Hiestand et al. (2004) find that self-paying (i.e., uninsured) patients with acute myocardial infarction (AMI)—one of the commonest diagnoses in our sample of nondeferrable conditions—are less likely to receive invasive treatments than those with insurance.

2In Card, Dobkin, and Maestas (2008) we show that there are large increases (10–20%) in rates of coronary bypass surgery, cholecystectomy (gall bladder removal), and many other procedures at age 65. We argue there that the pattern of effects is consistent with there being fewer restrictions in Medicare than in many private insurance plans.

3Spouses of people who qualify are also qualified. U.S. citizens and legal aliens with at least five years of residency can also enroll in Medicare at age 65 by paying monthly premiums.

4See Autor and Duggan (2003) for a recent analysis of trends in DI. A very small number of people who need kidney dialysis are also eligible.

5Unfortunately the probe is only asked of people over 65, so there is no way to know the degree of underreporting of Medicare coverage among people less than 65. Recent data from the Current Population Survey, which includes a series of insurance questions, suggest that the Medicare coverage rate rises from 17% to 85% between ages 64 and 65.

6The probe questions in the 2005 NHIS suggest that overall coverage after age 65 is underestimated in the NHIS by two to three percentage points (Cohen and Martinez 2007, Table 3).

7Medicare Parts A and B include significant deductibles and require a co-insurance payment of 20% on many bills. Some individuals obtain supplementary coverage through a previous employer, whereas others purchase private “Medigap” policies or enroll in Medicare-managed care plans.

8In our NHIS sample, about 85% of Medicare recipients are enrolled in traditional fee-for-service Medicare. Prior to 2003, the only managed care option in Medicare was to enroll in a Medicare HMO plan.

9Currie and Gruber (1996a, 1996b) find that Medicaid insurance for low-income pregnant women leads to improvements in health of newborns and a reduction in infant mortality.

10An exception is Card, Dobkin, and Maestas (2004), where we look at age profiles of self-reported health status. These are relatively smooth around age 65. Decker (2002) examines outcomes for breast cancer patients before and after Medicare eligibility and finds some evidence of better outcomes for those over 65.

11For example, in a randomized trial in which Medicare was made available to a treatment group of 65-year-olds and withheld from the controls, the program would have to have a 7% impact on annual mortality to yield a t-statistic of 2 or higher on the difference in one-year mortality between the treatment and control groups, even with 100,000 observations in each group. The reason is that the baseline mortality rate of 65-year-olds is only about 1.5% per year.

12See Imbens and Lemieux (2008) for an overview of recent work on RD methods. The causal effect is only identified for the subset of people whose status is changed at age 65.

13Arguably, one could break this effect out into an effect associated with Medicare coverage per se and an effect associated with coverage by multiple policies.

14Hospital admissions are typically much lower on weekends than weekdays, in part because of staffing constraints. Dobkin (2003) shows that mortality rates for patients admitted on the weekend for diagnoses with a constant daily admission rate are the same as those for patients admitted during the week.

15This figure shows employment rates by quarter of age, using data from the 1992–2003 National Health Interview Surveys. The spike in retirement at age 65 has largely disappeared in the past two decades (von Wachter 2002), reflecting the elimination of mandatory retirement and the availability of Social Security benefits at age 62.

16According to a national survey of hospitals conducted by the General Accounting Office (2003), approximately 15% of the patients seen in an ED are admitted to the hospital.

17This is defined as “the condition established, after study, to be the chief cause of the admission of the patient.”

18This restriction was imposed by the California Department of Health and Human Services as a condition for access to the discharge files.

19For all six models presented in the even-numbered columns of Table II the t-statistic for a test that the coefficient of the dummy is 0 is well below the usual critical value.

20We are grateful to a referee for suggesting this analysis. The Charlson comorbidity index is a weighted count of the presence of 19 diseases (e.g., diabetes with end organ damage is weighted 2, whereas peptic ulcer disease is weighted 1). We use the STATA coding for the index developed by Stagg (2006).

21We are grateful to a referee for suggesting that we consider this literature.

22Unfortunately we have no information on secondary coverage. We suspect that many of the 45% who have private coverage prior to age 65 enroll in Medicare and a supplementary policy at 65.

23We sum the duration of stay, list charges, and number of procedures for all consecutive stays. List charges are accounting charges, and do not represent the charges actually billed to insurers or patients. They also exclude charges for physician services, and are not reported for patients at Kaiser hospitals (hence the smaller sample size in columns (5)–(6) in Table IV). We interpret list charges as a convenient “price-weighted” summary of services rendered, albeit at artificial prices. Note that if list prices are a markup over actual costs, then the percentage change in list charges will be a good indicator of the change in the costs of the services provided to patients who are just under or just over 65.

24Cutler and McClellan (2001) have estimated that invasive diagnosis and treatment procedures as a whole (including catheterization, angioplasty, and bypass surgery) are cost-effective in the treatment of AMI. The efficacy of specific procedures is less clear: see, for example, McClellan, McNeil, and Newhouse (1994) and Cutler, McClellan, and Newhouse (1999).

25Note that to avoid double counting we have collapsed all consecutive hospital stays to single records.

26Here we follow the recommendation of Imbens and Lemieux (2008) and use a rectangular kernel for the local linear regressions on each side.

27To see that the effect of a small amount of sorting is negligible, note that even if (contrary to fact) the mortality rate at county hospitals were 50% higher than that of private hospitals, it could account for at most a negligible amount of the estimated mortality gain: 0.03 × 0.045 = 0.00135 percentage points.

28If α is the share of all potential patients who are only admitted after age 65, then the proportional increase in admissions at age 65 is (1 − (1 − α))/(1 − α) = α/(1 − α), so the RD in log admissions is an estimate of α/(1 − α).

29To construct a standard error for our lower bound we need to construct a standard error for Δ + α/(1 − α)m̄, where Δ is the estimated RD in mortality, α/(1 − α) is the estimated RD in log admissions, and m̄ is the estimated mortality rate for those just over 65, which can be estimated as the constant in the mortality regression (assuming age is normalized to 0 at age 65) plus the value of the RD in mortality. Because we need the covariance between the estimated parameters from the mortality and admissions models, we fit the two RDs as seemingly unrelated regressions using grouped age cells and use the delta method to construct the sampling error.

30We believe that any such data problems are likely to bias the results in the opposite direction. In particular, because the in-hospital mortality rate of people without SSNs is higher, at worst we would add to the sample at 65 a small group with higher potential mortality, which would lead to a rise in the measured death rate for people over 65.

31This equation simplifies health care services to a single dimension. In fact, changes in insurance can cause the use of some types of services to rise and the use of other types of services to fall (or stay constant).

32In particular, assume that π varies by subgroup, with a value of π (g) for subgroup g. If the parameters λ and θ are constant across groups then the discontinuity in services for group g is θπ (g) and the discontinuity in mortality is λθπ (g). By comparing the relative sizes of the discontinuities in insurance, treatment intensity, and mortality across subgroups it is possible to judge whether the data are consistent with a “1-channel” explanation.

33This net effect is likely a mix of some people attaining more generous coverage and others receiving less generous coverage relative to their pre-65 insurance plans. Even if Medicare is more generous with respect to case review procedures, it may be less generous on other dimensions, such as prescription drug coverage.

34At the suggestion of a referee we looked at differences in the magnitude of the changes in treatment intensity at age 65 for patients from California counties with relatively high rates of managed care among nonelderly patients, versus patients from counties with relatively low rates of managed care. Specifically, we used data from the 1998 Area Resource File to split counties in two groups, based on whether the fraction of non-Medicare patients in HMOs was under or over 44% (the HMO penetration rate of the median individual’s county). A specification parallel to the RD model in column (6) of Table IV yields an estimated jump in log list charges of 3.6% (standard error 1.8%) for counties with above-median HMO penetration, and 2.4% (standard error 1.1%) for counties with below-median HMO penetration. Models for the RDs in the number of procedures show the opposite pattern across the two groups of counties, but the difference is not statistically significant.

*We are extremely grateful to the editor and four referees for comments and suggestions on an earlier draft. We also thank the California Department of Health Services for providing the data used in the paper. This research was supported by the National Institute on Aging through Grant 1 R01 AG026290-01A1. The contents of this paper are solely the responsibility of the authors and do not necessarily represent the official views of the National Institute on Aging.

Contributor Information

David Card, University of California, Berkeley.

Carlos Dobkin, University of California, Santa Cruz.

Nicole Maestas, Rand Corporation.

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